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At the same time these astrocytes reduce expression of several key homoeostatic proteins such as glutamate transporters, glutamine synthetase, K ir 4.1 inward rectifying channels involved in K + buffering, and connexion 43 responsible for syncytial coupling

Participants with T2D described improved blood glucose control and reduction in HbA1c values at medical appointments

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[40] reported that exendin-4 increased LXR-mediated ABCA1 expression through AMPK activation, improving cholesterol-induced toxicity in INS-1 cells

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