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Dl-3- n -butylphthalide enhances collateral circulation by promoting endothelial nitric oxide synthase activity and activates neurotrophic signaling pathways such as brain-derived neurotrophic factor/tropomyosin receptor kinase B (TrkB) and calcitonin gene-related peptide (CGRP), thereby improving synaptic plasticity and cognitive recovery 17 (Fig

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Chondrocyte dedifferentiation was majorly characterized as the reduced expression of COL2 and ACAN and enhanced expression of type I collagen and versican ( COL2 expression, but not ACAN ( In addition to dedifferentiation, another consequence of in vitro expansion is replicative senescence, which also significantly influences chondrocyte function