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Failure to absorb cobalamin in dogs with EPI may be due to all three potential causes of low serum cobalamin
The potential metabolism-immune-neuro interaction framework of PEM is based on multi-level molecular crosstalk: mitochondrial dysfunction generates DAMPs that activate innate immune pathways (NLRP3, NF-B) to induce peripheral inflammation
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These effects support the stability of calcium signaling and homeostasis and indicate a treatment-induced neuroprotective effect against apoptosis, which is in line with previous studies showing a decrease in hippocampal apoptotic cell death and caspase activation caused by supplementation with N-3 PUFAs in aged rats, and by Vit D in rats exposed to other neurotoxic agents 84,85,86
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