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Although there appears to be a high degree of overlap in GLP-1R expression in the rodent and non-human primate brain, one notable difference may be an apparent higher expression of GLP-1R in the central amygdala and bed nucleus of the stria terminalis of non-human primates (86)

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Yet, a small fraction of patients remained on the starting 3 mg dose, which is not considered a maintenance dose of oral semaglutide

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however, neither inactivation of KCs nor neutralization of tumor necrosis factor (TNF) reversed ethanol-induced suppression of hepcidin expression, indicating that KCs are not involved in the decline of hepcidin expression in ALD [89]