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Antioxidant Depletion: In an ironnitrilotriacetic acid rat model of hemochromatosis, renal samples showed increases in reactive intermediates, accompanied by decreased availability of systems that protect against oxidation by iron, such as vitamin E levels, the glutathione system, thiol-specific antioxidants, and superoxide dismutase

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Moreover, treatment with GLP-1 resulted in a decrease of lipopolysaccharide (LPS)-induced activation of the BV-2 microglial cell line, accompanied by a decrease in TNF- secretion [63]

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