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As superoxide is subject to efficient dismutation by cellular superoxide dismutases, it is possible that NFT might directly interact with Keap1 to induce the observed release of Nrf2 and its escape from proteasomal degradation

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Glutathione helps neutralize these radicals by converting them into harmless compounds, protecting muscle and liver cells in the process

Interaction between TAK1-TAB1-TAB2 and RCAN1-calcineurin defines a signalling nodal control point