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Modern incretin-based therapy still relies heavily on appetite suppression, but the addition of glucagon receptor activation may shift the model toward a broader metabolic strategy: reducing food intake while increasing energy expenditure and improving hepatic lipid metabolism

137 In addition, adipose MSCs, hair follicle MSCs, and dental pulp MSCs reduce inflammatory responses, promote mucosal healing, and improve intestinal barrier function

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This cellular protection supports muscle recovery and reduces prolonged inflammatory signaling that can interfere with performance and healing