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Without coenzyme B12, tetrahydrofolate cannot be produced from 5-methyl tetrahydrofolate leading to folate deficiency

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Heteroatom doping, surface functionalisation, and composite formation enhance electrostatic interactions with microbial membranes, promote ROS generation, and enable targeted disruption of viral surface proteins or microbial cell walls

At the same time, B12 insufficiency in the liver leads to shrinking of the intestinal cells and malabsorption of nutrients (including B12 itself), leading to a cycle of worsening B12 deficiency

Although early research is encouraging, clinical evidence in humans remains limited and further studies are needed