The SSRI, which inhibit serotonin transporter SERT/SLC6A4 (expressed in both neurones and astroglia) and thus increase bioavailability of serotonin, are probably the most widely used in the treatment of depression
and I don't understand the mechanism here, but I've heard this anecdotally, and maybe you can explain it to me
The ability of AtGLRs to form diverse oligomeric assemblies likely contributes to their responsiveness to a broad spectrum of metabolite-derived ligands, including amino acids, organic acids, and secondary metabolites, thereby expanding their signalling flexibility
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