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The Regulation of Ferroptosis by Tumor Suppressor p53 and its Pathway

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The oxidant/antioxidant imbalance is not peculiar to SARS, but it is shared by all inflammatory lung diseases in which the activation of redox-sensitive transcription factors such as NF-kB is reduced by GSH [37]

[6] GH itself, when administered exogenously at doses above 0.03 mg/kg/day, has been associated with transient rises in ALT of approximately 10 to 20% above baseline in a subset of patients with pre-existing metabolic liver disease

Previous in vitro research has revealed that supplementation of GSH at a concentration of 5 mM to H