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In clinical studies, the stimulatory effect of glucagon on energy expenditure is heterogeneous depending on feeding status (preprandial vs postprandial) and the mechanism by which this occurs is not known

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Our previous studies in mice with a homozygotic deletion of Glud1 in CNS (C- Glud1 / mice) revealed elevated hippocampal cerebral blood volume (CBV) and glutamate levels in medial PFC (mPFC) and hippocampus, along with baseline and amphetamine-induced hyperlocomotion

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