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It is unknown whether suppressing the anti-cancer immune response generated by M2 macrophage polarization in the TME is more effective than boosting the anti-cancer immune response generated by M1 macrophage polarization because M2 macrophages consume more glutamine than M1 macrophages (Fig

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Preclinical studies have found that Ex-4 significantly upregulates Sirt3 expression in glioblastoma by activating the GLP-1R, which subsequently inhibits the growth, migration, invasion, and epithelial-mesenchymal transition of glioma cells [87]