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The short dihexa half life is a pharmacological advantage, not a limitation it allows precise temporal control over receptor activation without accumulation toxicity or prolonged systemic exposure

Involvement of mitochondria in acetaminophen-induced apoptosis and hepatic injury: Roles of cytochrome c, Bax, bid, and caspases

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Sluggish metabolism slowing your progress

It offers absolute quantification but requires sacrifice of multiple animals